To understand the relationship between depression and insomnia, let us first understand the individual meaning of both. Insomnia is a sleep disorder, where people find it difficult to sleep or stay asleep as long as desired. Insomnia mostly causes daytime sleepiness, irritation, and mood depression.
Depression is something when people feel upset and demotivated. this generally happens when people go through difficult times. It increases stress and anxiety, which hampers the normal activity of brain. One of the things that comes during depression is, people won’t be able to sleep easily. So, basically, insomnia is one of the symptoms of depression.
Depressive patients may have one of the following abnormal sleep types:
- Increased sleep time
- Reduced total sleep time
- Increased sleep with increased wake-up time after the onset of sleep
- Sleep latency
- Increase in paradoxical sleep time at the onset of the night in.
The relationship between depression and insomnia can be considered according to the time of onset of symptoms of each disorder. Any information that can be acquired on which disorder substitute or coincides with the other will play a role in the development of optimal treatment strategies.
Depression and insomnia, which comes first?
Depression and insomnia often go together. More than 80% of people have difficulty falling asleep or staying asleep. Patients suffering from persistent insomnia are more than three times more likely to develop depression. Insomnia and depression are two distinct but partly coinciding disorder. Some research shows that treating both the disorder simultaneously can improve the sleep quality, patient mood, and their overall life better.
It is possible to identify and differentiate insomnia related to depression and primary insomnia by identifying the particular several daily symptoms which are typical for depression, such as persistent depressed mood and suicidal tendency.
How Does Depression Affect Sleep
One of the first symptoms of depression is insomnia is they can not fall asleep and they can’t stay asleep. And then the question arises, how to sleep with depression and anxiety?
To get the answer, first the type of sleep should be known. There are times when you can almost diagnose depression by looking at individual sleep studies, where the structure of sleep becomes out of line:
- Deep sleep, where we produce growth hormone, a crucial step for memory and decision making, can completely disappear with depression.
- Awakening from sleep increase dramatically. Sleep becomes very fragmented.
- REM sleep is often broken and its appearance changes. Fast eye movements can appear both erratic and dense.
Can insomnia trigger depression?
When people can’t sleep properly, they become anxious, and afterward that anxiety results in depression. But the relationship is much more than just cause and effect. When depressed people suffer from insomnia, their risk of recovering from depression is higher than that of patients who do not suffer from insomnia. So in this way, insomnia may cause a serve for the trigger for depression.
Depression Causes prolonged sleepiness
How can a disease cause insomnia and its opposite? Try to think of depression as being what it is a massive distortion in the brain Information network. Some depressive people sleeps too much while others do not sleep much. Just as there are hundreds of causes of depression, its manifestations are multiple. People with the bipolar disease can demonstrate a more compressed version of this aberrant cerebral sleep control, sleeping two hours a night and sixteen hours the next day.
There is a complex relationship between lack of sleep and depression. The focus should be more on the treatment of lack of sleep or insomnia in depression because it is a risk factor for depression. Insomnia also plays an important role in the evolution and severity of the depressive mood, and persistent insomnia is a risk factor for worsening depression. Treatment strategies should address both depression symptoms and insomnia and should consider the use of pharmacological and non-pharmacological strategies.